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We examine the IASP definition of pain, why shared language matters, and how sloppy semantics derail research and care. We contrast barriers to recovery with causes, clarify differences between nociceptors and nociception, and use sensitization and nociplastic as hypotheses that guide better reasoning.

• using the IASP definition to ground assessment
• distinguishing nociceptors, neurons, and nociception
• reframing biopsychosocial as barriers to recovery
• identifying sensitization without overpathologizing
• applying nociceptive, nociplastic, neuropathic as hypotheses
• avoiding conflations like “pain signals” in clinic and AI
• aligning treatment dosing to irritability and mechanism

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